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We find that chronic induction of prc by steroid hormones stimulates excess PRC protein in the ECM surrounding the myocardial-nephrocyte cells, induces proteinuria and inhibits nephrocyte filtration, which contributes to its pathophysiology," the authors concluded. "This work reveals that low levels of prc, and the protein associated with the formation of the ECM, contribute to the pathophysiology of heart ventricular systole in young patients. Furthermore, the protein is also associated with the severity of ventricular tachycardia induced by treatment with synthetic hormone." They propose that an appropriate therapy for treatment of patients with chronic venous occlusion is to induce the formation of prc on a regular basis, with or without the inclusion of the presence of atrioventricular damage, since it appears that the latter in high-risk patients may promote the formation of prc. Explore further: New drug-based approach to venous occlusion prevention More information: E. A. Zafar et al. The Efficient Bioconjugate Formation of Pramlintide is Highly Enzyme-Activated and Contribute to the Pathophysiology of Venous Insufficiency, Journal of Clinical Investigation (2016). DOI: 10.1172/JCI292926 Similar articles:


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